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intact cell respiration  (Thermo Fisher)


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    Thermo Fisher intact cell respiration
    Tobacco smoke and E‐cig vapor condensates activate the AHR pathway and alter mitochondrial <t>respiration</t> in cultured muscle cells. (a) C2C12 myotubes were treated for 16 h without affecting cell viability. (b–d) mRNA expression of Ahr , Ahrr , and Cyp1a1 in treated myotubes. (e) Schematic overview of mitochondrial assay. (f, g) Mitochondrial respiration assessed using respirometry. Data analyzed using Kruskal‐Wallis test. N = 4–6 biological replicates per group. Error bars represent the standard deviation.
    Intact Cell Respiration, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/intact cell respiration/product/Thermo Fisher
    Average 97 stars, based on 1 article reviews
    intact cell respiration - by Bioz Stars, 2026-05
    97/100 stars

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    1) Product Images from "E‐cigarette exposure impairs skeletal muscle mitochondrial function in male mice"

    Article Title: E‐cigarette exposure impairs skeletal muscle mitochondrial function in male mice

    Journal: Physiological Reports

    doi: 10.14814/phy2.70797

    Tobacco smoke and E‐cig vapor condensates activate the AHR pathway and alter mitochondrial respiration in cultured muscle cells. (a) C2C12 myotubes were treated for 16 h without affecting cell viability. (b–d) mRNA expression of Ahr , Ahrr , and Cyp1a1 in treated myotubes. (e) Schematic overview of mitochondrial assay. (f, g) Mitochondrial respiration assessed using respirometry. Data analyzed using Kruskal‐Wallis test. N = 4–6 biological replicates per group. Error bars represent the standard deviation.
    Figure Legend Snippet: Tobacco smoke and E‐cig vapor condensates activate the AHR pathway and alter mitochondrial respiration in cultured muscle cells. (a) C2C12 myotubes were treated for 16 h without affecting cell viability. (b–d) mRNA expression of Ahr , Ahrr , and Cyp1a1 in treated myotubes. (e) Schematic overview of mitochondrial assay. (f, g) Mitochondrial respiration assessed using respirometry. Data analyzed using Kruskal‐Wallis test. N = 4–6 biological replicates per group. Error bars represent the standard deviation.

    Techniques Used: Cell Culture, Expressing, Standard Deviation

    Chronic tobacco smoke or E‐cig exposure reduces skeletal muscle mitochondrial respiration but not contractile function in male mice. (a) Graphical depiction of experimental groups and exposure time. (b) Body weight. (c) Mass of hindlimb muscles normalized by body weight. (d) Schematic overview of mitochondrial assay. (e–h) Mitochondrial respiratory function. (i) Schematic overview of in situ muscle function testing. (j–k) Plantar flexor muscle contractile strength measured in situ. Data analyzed using Kruskal–Wallis test. N = 4 per group. Error bars represent the standard deviation.
    Figure Legend Snippet: Chronic tobacco smoke or E‐cig exposure reduces skeletal muscle mitochondrial respiration but not contractile function in male mice. (a) Graphical depiction of experimental groups and exposure time. (b) Body weight. (c) Mass of hindlimb muscles normalized by body weight. (d) Schematic overview of mitochondrial assay. (e–h) Mitochondrial respiratory function. (i) Schematic overview of in situ muscle function testing. (j–k) Plantar flexor muscle contractile strength measured in situ. Data analyzed using Kruskal–Wallis test. N = 4 per group. Error bars represent the standard deviation.

    Techniques Used: Muscles, In Situ, Standard Deviation



    Similar Products

    intact cell respiration  (Thermo Fisher)
    97
    Thermo Fisher intact cell respiration
    Tobacco smoke and E‐cig vapor condensates activate the AHR pathway and alter mitochondrial <t>respiration</t> in cultured muscle cells. (a) C2C12 myotubes were treated for 16 h without affecting cell viability. (b–d) mRNA expression of Ahr , Ahrr , and Cyp1a1 in treated myotubes. (e) Schematic overview of mitochondrial assay. (f, g) Mitochondrial respiration assessed using respirometry. Data analyzed using Kruskal‐Wallis test. N = 4–6 biological replicates per group. Error bars represent the standard deviation.
    Intact Cell Respiration, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/intact cell respiration/product/Thermo Fisher
    Average 97 stars, based on 1 article reviews
    intact cell respiration - by Bioz Stars, 2026-05
    97/100 stars
    		  Buy from Supplier

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    Tobacco smoke and E‐cig vapor condensates activate the AHR pathway and alter mitochondrial respiration in cultured muscle cells. (a) C2C12 myotubes were treated for 16 h without affecting cell viability. (b–d) mRNA expression of Ahr , Ahrr , and Cyp1a1 in treated myotubes. (e) Schematic overview of mitochondrial assay. (f, g) Mitochondrial respiration assessed using respirometry. Data analyzed using Kruskal‐Wallis test. N = 4–6 biological replicates per group. Error bars represent the standard deviation.

    Journal: Physiological Reports

    Article Title: E‐cigarette exposure impairs skeletal muscle mitochondrial function in male mice

    doi: 10.14814/phy2.70797

    Figure Lengend Snippet: Tobacco smoke and E‐cig vapor condensates activate the AHR pathway and alter mitochondrial respiration in cultured muscle cells. (a) C2C12 myotubes were treated for 16 h without affecting cell viability. (b–d) mRNA expression of Ahr , Ahrr , and Cyp1a1 in treated myotubes. (e) Schematic overview of mitochondrial assay. (f, g) Mitochondrial respiration assessed using respirometry. Data analyzed using Kruskal‐Wallis test. N = 4–6 biological replicates per group. Error bars represent the standard deviation.

    Article Snippet: First, intact cell respiration was measured, followed by the addition of digitonin (10 μg/mL; ThermoFisher Scientific; Cat. No. BN2006) to permeabilize the myotubes.

    Techniques: Cell Culture, Expressing, Standard Deviation

    Chronic tobacco smoke or E‐cig exposure reduces skeletal muscle mitochondrial respiration but not contractile function in male mice. (a) Graphical depiction of experimental groups and exposure time. (b) Body weight. (c) Mass of hindlimb muscles normalized by body weight. (d) Schematic overview of mitochondrial assay. (e–h) Mitochondrial respiratory function. (i) Schematic overview of in situ muscle function testing. (j–k) Plantar flexor muscle contractile strength measured in situ. Data analyzed using Kruskal–Wallis test. N = 4 per group. Error bars represent the standard deviation.

    Journal: Physiological Reports

    Article Title: E‐cigarette exposure impairs skeletal muscle mitochondrial function in male mice

    doi: 10.14814/phy2.70797

    Figure Lengend Snippet: Chronic tobacco smoke or E‐cig exposure reduces skeletal muscle mitochondrial respiration but not contractile function in male mice. (a) Graphical depiction of experimental groups and exposure time. (b) Body weight. (c) Mass of hindlimb muscles normalized by body weight. (d) Schematic overview of mitochondrial assay. (e–h) Mitochondrial respiratory function. (i) Schematic overview of in situ muscle function testing. (j–k) Plantar flexor muscle contractile strength measured in situ. Data analyzed using Kruskal–Wallis test. N = 4 per group. Error bars represent the standard deviation.

    Article Snippet: First, intact cell respiration was measured, followed by the addition of digitonin (10 μg/mL; ThermoFisher Scientific; Cat. No. BN2006) to permeabilize the myotubes.

    Techniques: Muscles, In Situ, Standard Deviation